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Seizure activity results in calcium- and mitochondria-independent ROS production via NADPH and xanthine oxidase activation
Seizure activity has been proposed to result in the generation of reactive oxygen species (ROS), which then contribute to seizure-induced neuronal damage and eventually cell death. Although the mechanisms of seizure-induced ROS generation are unclear, mitochondria and cellular calcium overload have...
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| Pubblicato in: | Cell Death Dis |
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| Autori principali: | , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Nature Publishing Group
2014
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4649505/ https://ncbi.nlm.nih.gov/pubmed/25275601 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2014.390 |
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