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O-GlcNAcase overexpression reverses coronary endothelial cell dysfunction in type 1 diabetic mice
Cardiovascular disease is the primary cause of morbidity and mortality in diabetes, and endothelial dysfunction is commonly seen in these patients. Increased O-linked N-acetylglucosamine (O-GlcNAc) protein modification is one of the central pathogenic features of diabetes. Modification of proteins b...
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| Опубликовано в: : | Am J Physiol Cell Physiol |
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| Главные авторы: | , , , , , , , , |
| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
American Physiological Society
2015
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4628934/ https://ncbi.nlm.nih.gov/pubmed/26269457 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00069.2015 |
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