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Suppression of the alternative lengthening of telomere pathway by the chromatin remodelling factor ATRX
Fifteen per cent of cancers maintain telomere length independently of telomerase by the homologous recombination (HR)-associated alternative lengthening of telomeres (ALT) pathway. A unifying feature of these tumours are mutations in ATRX. Here we show that expression of ectopic ATRX triggers a supp...
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| 出版年: | Nat Commun |
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| 主要な著者: | , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Nature Pub. Group
2015
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4501375/ https://ncbi.nlm.nih.gov/pubmed/26143912 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ncomms8538 |
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