OXIDATIVE INHIBITION OF THE VASCULAR Na(+)-K(+) PUMP VIA NADPH OXIDASE-DEPENDENT β(1) SUBUNIT GLUTATHIONYLATION: IMPLICATIONS FOR ANGIOTENSIN II-INDUCED VASCULAR DYSFUNCTION

Documenti analoghi

• Glutathionylation Mediates Angiotensin II–Induced eNOS Uncoupling, Amplifying NADPH Oxidase‐Dependent Endothelial Dysfunction
  di: Galougahi, Keyvan Karimi, et al.
  Pubblicazione: (2014)
• Susceptibility of β1 Na(+)-K(+) Pump Subunit to Glutathionylation and Oxidative Inhibition Depends on Conformational State of Pump
  di: Liu, Chia-Chi, et al.
  Pubblicazione: (2012)
• Protein kinase-dependent oxidative regulation of the cardiac Na(+)–K(+) pump: evidence from in vivo and in vitro modulation of cell signalling
  di: Galougahi, Keyvan Karimi, et al.
  Pubblicazione: (2013)
• β(3)-Adrenoceptor activation relieves oxidative inhibition of the cardiac Na(+)-K(+) pump in hyperglycemia induced by insulin receptor blockade
  di: Karimi Galougahi, Keyvan, et al.
  Pubblicazione: (2015)
• FXYD Proteins Reverse Inhibition of the Na(+)-K(+) Pump Mediated by Glutathionylation of Its β(1) Subunit
  di: Bibert, Stéphanie, et al.
  Pubblicazione: (2011)