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Neutralizing the EGF receptor in glioblastoma cells stimulates cell migration by activating uPAR-initiated cell-signaling
In glioblastoma (GBM), the EGF receptor (EGFR) and Src family kinases (SFKs) contribute to an aggressive phenotype. EGFR may be targeted therapeutically; however, resistance to EGFR-targeting drugs such as Erlotinib and Gefitinib develops quickly. In many GBMs, a truncated form of the EGFR (EGFRvIII...
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| Publicado no: | Oncogene |
|---|---|
| Main Authors: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4411189/ https://ncbi.nlm.nih.gov/pubmed/25347738 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/onc.2014.336 |
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