CaM Kinase II mediates maladaptive post-infarct remodeling and pro-inflammatory chemoattractant signaling but not acute myocardial ischemia/reperfusion injury

CaMKII was suggested to mediate ischemic myocardial injury and adverse cardiac remodeling. Here, we investigated the roles of different CaMKII isoforms and splice variants in ischemia/reperfusion (I/R) injury by the use of new genetic CaMKII mouse models. Although CaMKIIδC was upregulated 1 day afte...

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Bibliografski detalji
Izdano u:EMBO Mol Med
Glavni autori: Weinreuter, Martin, Kreusser, Michael M, Beckendorf, Jan, Schreiter, Friederike C, Leuschner, Florian, Lehmann, Lorenz H, Hofmann, Kai P, Rostosky, Julia S, Diemert, Nathalie, Xu, Chang, Volz, Hans Christian, Jungmann, Andreas, Nickel, Alexander, Sticht, Carsten, Gretz, Norbert, Maack, Christoph, Schneider, Michael D, Gröne, Hermann-Josef, Müller, Oliver J, Katus, Hugo A, Backs, Johannes
Format: Artigo
Jezik:Inglês
Izdano: BlackWell Publishing Ltd 2014
Teme:
Research Articles
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC4287929/
https://ncbi.nlm.nih.gov/pubmed/25193973
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/emmm.201403848
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