CaM Kinase II mediates maladaptive post-infarct remodeling and pro-inflammatory chemoattractant signaling but not acute myocardial ischemia/reperfusion injury

CaMKII was suggested to mediate ischemic myocardial injury and adverse cardiac remodeling. Here, we investigated the roles of different CaMKII isoforms and splice variants in ischemia/reperfusion (I/R) injury by the use of new genetic CaMKII mouse models. Although CaMKIIδC was upregulated 1 day afte...

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Publicat a:EMBO Mol Med
Autors principals: Weinreuter, Martin, Kreusser, Michael M, Beckendorf, Jan, Schreiter, Friederike C, Leuschner, Florian, Lehmann, Lorenz H, Hofmann, Kai P, Rostosky, Julia S, Diemert, Nathalie, Xu, Chang, Volz, Hans Christian, Jungmann, Andreas, Nickel, Alexander, Sticht, Carsten, Gretz, Norbert, Maack, Christoph, Schneider, Michael D, Gröne, Hermann-Josef, Müller, Oliver J, Katus, Hugo A, Backs, Johannes
Format: Artigo
Idioma:Inglês
Publicat: BlackWell Publishing Ltd 2014
Matèries:
Research Articles
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4287929/
https://ncbi.nlm.nih.gov/pubmed/25193973
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/emmm.201403848
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