CaM Kinase II mediates maladaptive post-infarct remodeling and pro-inflammatory chemoattractant signaling but not acute myocardial ischemia/reperfusion injury

CaMKII was suggested to mediate ischemic myocardial injury and adverse cardiac remodeling. Here, we investigated the roles of different CaMKII isoforms and splice variants in ischemia/reperfusion (I/R) injury by the use of new genetic CaMKII mouse models. Although CaMKIIδC was upregulated 1 day afte...

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Опубликовано в: :EMBO Mol Med
Главные авторы: Weinreuter, Martin, Kreusser, Michael M, Beckendorf, Jan, Schreiter, Friederike C, Leuschner, Florian, Lehmann, Lorenz H, Hofmann, Kai P, Rostosky, Julia S, Diemert, Nathalie, Xu, Chang, Volz, Hans Christian, Jungmann, Andreas, Nickel, Alexander, Sticht, Carsten, Gretz, Norbert, Maack, Christoph, Schneider, Michael D, Gröne, Hermann-Josef, Müller, Oliver J, Katus, Hugo A, Backs, Johannes
Формат: Artigo
Язык:Inglês
Опубликовано: BlackWell Publishing Ltd 2014
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Research Articles
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC4287929/
https://ncbi.nlm.nih.gov/pubmed/25193973
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/emmm.201403848
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