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The Q510E mutation in Shp2 perturbs heart valve development by increasing cell migration

Tightly regulated cellular signaling is critical for correct heart valve development, but how and why signaling is dysregulated in congenital heart disease is not very well known. We focused on protein tyrosine phosphatase Shp2, because mutations in this signaling modulator frequently cause valve ma...

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Dades bibliogràfiques
Publicat a:J Appl Physiol (1985)
Autors principals: Edwards, Michelle A., Crombie, Kathryn, Schramm, Christine, Krenz, Maike
Format: Artigo
Idioma:Inglês
Publicat: American Physiological Society 2014
Matèries:
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4281644/
https://ncbi.nlm.nih.gov/pubmed/25359717
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/japplphysiol.00008.2014
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