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New Approaches to Prevent LEOPARD Syndrome-associated Cardiac Hypertrophy by Specifically Targeting Shp2-dependent Signaling

In LEOPARD syndrome (LS) patients, mutations in the protein tyrosine phosphatase Shp2 cause hypertrophic cardiomyopathy. The prohypertrophic effects of mutant Shp2 are mediated downstream by hyperactivation of mammalian target of rapamycin. Our goal was to further define the signaling cascade that i...

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Bibliografische gegevens
Hoofdauteurs: Schramm, Christine, Edwards, Michelle A., Krenz, Maike
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: American Society for Biochemistry and Molecular Biology 2013
Onderwerpen:
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3689975/
https://ncbi.nlm.nih.gov/pubmed/23673659
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M113.483800
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