The Extracellular Adherence Protein from Staphylococcus aureus Inhibits the Classical and Lectin Pathways of Complement by Blocking Formation of the C3 Pro-Convertase

The pathogenic bacterium Staphylococcus aureus actively evades many aspects of human innate immunity by expressing a series of small inhibitory proteins. A number of these proteins inhibit the complement system, which labels bacteria for phagocytosis and generates inflammatory chemoattractants. Whil...

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Bibliografiset tiedot
Julkaisussa:J Immunol
Päätekijät: Woehl, Jordan L., Stapels, Daphne A.C., Garcia, Brandon L., Ramyar, Kasra X., Keightley, Andrew, Ruyken, Maartje, Syriga, Maria, Sfyroera, Georgia, Weber, Alexander B., Zolkiewski, Michal, Ricklin, Daniel, Lambris, John D., Rooijakkers, Suzan H.M., Geisbrecht, Brian V.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2014
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Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4258549/
https://ncbi.nlm.nih.gov/pubmed/25381436
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1401600
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