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Antigen-specific TCR–pMHC catch bonds trigger signaling by fast accumulation of force-prolonged bond lifetimes

TCR–pMHC interactions initiate adaptive immune responses, but the mechanism of how such interactions under force induce T-cell signaling is unclear. We show that force prolongs lifetimes of single TCR–pMHC bonds for agonists (catch bonds) but shortens those for antagonists (slip bonds). Both magnitu...

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Autori principali: Liu, Baoyu, Chen, Wei, Evavold, Brian D., Zhu, Cheng
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2014
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4123688/
https://ncbi.nlm.nih.gov/pubmed/24725404
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cell.2014.02.053
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