β-Adrenergic receptor blockade arrests myocyte damage and preserves cardiac function in the transgenic G(sα) mouse

Transgenic (TG) mice with cardiac G(sα) overexpression exhibit enhanced inotropic and chronotropic responses to sympathetic stimulation, but develop cardiomyopathy with age. We tested the hypothesis that cardiomyopathy in TG mice with G(sα) overexpression could be averted with chronic β-adrenergic r...

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Detalhes bibliográficos
Main Authors: Asai, Kuniya, Yang, Gui-Ping, Geng, Yong-Jian, Takagi, Gen, Bishop, Sanford, Ishikawa, Yoshihiro, Shannon, Richard P., Wagner, Thomas E., Vatner, Dorothy E., Homcy, Charles J., Vatner, Stephen F.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Clinical Investigation 1999
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC408547/
https://ncbi.nlm.nih.gov/pubmed/10487769
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