Nitrosation-dependent caveolin 1 phosphorylation, ubiquitination, and degradation and its association with idiopathic pulmonary arterial hypertension

Abstract. In the present study, we tested the hypothesis that chronic inflammation and oxidative/nitrosative stress induce caveolin 1 (Cav-1) degradation, providing an underlying mechanism of endothelial cell activation/dysfunction and pulmonary vascular remodeling in patients with idiopathic pulmon...

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Autors principals: Bakhshi, Farnaz R., Mao, Mao, Shajahan, Ayesha N., Piegeler, Tobias, Chen, Zhenlong, Chernaya, Olga, Sharma, Tiffany, Elliott, W. Mark, Szulcek, Robert, Bogaard, Harm Jan, Comhair, Suzy, Erzurum, Serpil, van Nieuw Amerongen, Geerten P., Bonini, Marcelo G., Minshall, Richard D.
Format: Artigo
Idioma:Inglês
Publicat: University of Chicago Press 2013
Matèries:
Original Research
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4070841/
https://ncbi.nlm.nih.gov/pubmed/25006397
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1086/674753
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