Toll-Like Receptor 4–Induced IL-22 Accelerates Kidney Regeneration

AKI involves early Toll-like receptor (TLR)–driven immunopathology, and resolution of inflammation is needed for rapid regeneration of injured tubule cells. Notably, activation of TLRs also has been implicated in epithelial repair. We hypothesized that TLR signaling drives tubule regeneration after...

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Autori principali: Kulkarni, Onkar P., Hartter, Ingo, Mulay, Shrikant R., Hagemann, Jan, Darisipudi, Murthy N., Kumar VR, Santhosh, Romoli, Simone, Thomasova, Dana, Ryu, Mi, Kobold, Sebastian, Anders, Hans-Joachim
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Society of Nephrology 2014
Soggetti:
Basic Research
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4005301/
https://ncbi.nlm.nih.gov/pubmed/24459235
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2013050528
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