Toll-Like Receptor 4–Induced IL-22 Accelerates Kidney Regeneration

AKI involves early Toll-like receptor (TLR)–driven immunopathology, and resolution of inflammation is needed for rapid regeneration of injured tubule cells. Notably, activation of TLRs also has been implicated in epithelial repair. We hypothesized that TLR signaling drives tubule regeneration after...

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Главные авторы: Kulkarni, Onkar P., Hartter, Ingo, Mulay, Shrikant R., Hagemann, Jan, Darisipudi, Murthy N., Kumar VR, Santhosh, Romoli, Simone, Thomasova, Dana, Ryu, Mi, Kobold, Sebastian, Anders, Hans-Joachim
Формат: Artigo
Язык:Inglês
Опубликовано: American Society of Nephrology 2014
Предметы:
Basic Research
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC4005301/
https://ncbi.nlm.nih.gov/pubmed/24459235
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2013050528
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