Toll-Like Receptor 4–Induced IL-22 Accelerates Kidney Regeneration

AKI involves early Toll-like receptor (TLR)–driven immunopathology, and resolution of inflammation is needed for rapid regeneration of injured tubule cells. Notably, activation of TLRs also has been implicated in epithelial repair. We hypothesized that TLR signaling drives tubule regeneration after...

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Bibliografische gegevens
Hoofdauteurs: Kulkarni, Onkar P., Hartter, Ingo, Mulay, Shrikant R., Hagemann, Jan, Darisipudi, Murthy N., Kumar VR, Santhosh, Romoli, Simone, Thomasova, Dana, Ryu, Mi, Kobold, Sebastian, Anders, Hans-Joachim
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: American Society of Nephrology 2014
Onderwerpen:
Basic Research
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4005301/
https://ncbi.nlm.nih.gov/pubmed/24459235
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2013050528
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