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Toll-Like Receptor 4–Induced IL-22 Accelerates Kidney Regeneration
AKI involves early Toll-like receptor (TLR)–driven immunopathology, and resolution of inflammation is needed for rapid regeneration of injured tubule cells. Notably, activation of TLRs also has been implicated in epithelial repair. We hypothesized that TLR signaling drives tubule regeneration after...
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| Главные авторы: | , , , , , , , , , , |
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| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
American Society of Nephrology
2014
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4005301/ https://ncbi.nlm.nih.gov/pubmed/24459235 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2013050528 |
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