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CHBPR: ENDOPLASMIC RETICULUM STRESS CONTRIBUTES TO AORTIC STIFFENING VIA PRO-APOPTOTIC AND FIBROTIC SIGNALING MECHANISMS
Vascular smooth muscle cell (VSMC) apoptosis and collagen synthesis contributes to aortic stiffening. A cellular signaling mechanism contributing to apoptotic and fibrotic events is endoplasmic reticulum (ER) stress. In this study we tested the hypothesis that induction of ER stress in a normotensiv...
में बचाया:
| मुख्य लेखकों: | , |
|---|---|
| स्वरूप: | Artigo |
| भाषा: | Inglês |
| प्रकाशित: |
2013
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| विषय: | |
| ऑनलाइन पहुंच: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4004348/ https://ncbi.nlm.nih.gov/pubmed/24379182 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/HYPERTENSIONAHA.113.02558 |
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