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CHBPR: ENDOPLASMIC RETICULUM STRESS CONTRIBUTES TO AORTIC STIFFENING VIA PRO-APOPTOTIC AND FIBROTIC SIGNALING MECHANISMS

Vascular smooth muscle cell (VSMC) apoptosis and collagen synthesis contributes to aortic stiffening. A cellular signaling mechanism contributing to apoptotic and fibrotic events is endoplasmic reticulum (ER) stress. In this study we tested the hypothesis that induction of ER stress in a normotensiv...

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Detalhes bibliográficos
Main Authors: Spitler, Kathryn M., Webb, R. Clinton
Formato: Artigo
Idioma:Inglês
Publicado em: 2013
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4004348/
https://ncbi.nlm.nih.gov/pubmed/24379182
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/HYPERTENSIONAHA.113.02558
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