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Is Alzheimer’s disease amyloidosis the result of a repair mechanism gone astray?
We synthesize several lines of evidence supporting the hypothesis that at least one function of Aβ is to serve as a part of the acute response to brain hemodynamic disturbances intended to seal vascular leakage. Given the resilient and adhesive physicochemical properties of amyloid, an abluminal hem...
Gorde:
| Egile Nagusiak: | , , |
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| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
2011
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3984046/ https://ncbi.nlm.nih.gov/pubmed/22047632 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jalz.2011.05.2429 |
| Etiketak: |
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