Prolactin-Stat5 signaling in breast cancer is potently disrupted by acidosis within the tumor microenvironment

INTRODUCTION: Emerging evidence in estrogen receptor-positive breast cancer supports the notion that prolactin-Stat5 signaling promotes survival and maintenance of differentiated luminal cells, and loss of nuclear tyrosine phosphorylated Stat5 (Nuc-pYStat5) in clinical breast cancer is associated wi...

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Hlavní autoři: Yang, Ning, Liu, Chengbao, Peck, Amy R, Girondo, Melanie A, Yanac, Alicia F, Tran, Thai H, Utama, Fransiscus E, Tanaka, Takemi, Freydin, Boris, Chervoneva, Inna, Hyslop, Terry, Kovatich, Albert J, Hooke, Jeffrey A, Shriver, Craig D, Rui, Hallgeir
Médium: Artigo
Jazyk:Inglês
Vydáno: BioMed Central 2013
Témata:
Research Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3978581/
https://ncbi.nlm.nih.gov/pubmed/24004716
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/bcr3467
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