Prolactin suppresses a progestin-induced CK5-positive cell population in luminal breast cancer through inhibition of progestin-driven BCL6 expression

Prolactin controls the development and function of milk-producing breast epithelia but also supports growth and differentiation of breast cancer, especially luminal subtypes. A principal signaling mediator of prolactin, Stat5, promotes cellular differentiation of breast cancer cells in vitro, and lo...

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מידע ביבליוגרפי
Main Authors: Sato, Takahiro, Tran, Thai H., Peck, Amy R., Girondo, Melanie A., Liu, Chengbao, Goodman, Chelain R., Neilson, Lynn M., Freydin, Boris, Chervoneva, Inna, Hyslop, Terry, Kovatich, Albert J., Hooke, Jeffrey A., Shriver, Craig D., Fuchs, Serge Y., Rui, Hallgeir
פורמט: Artigo
שפה:Inglês
יצא לאור: 2013
נושאים:
Article
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC3913798/
https://ncbi.nlm.nih.gov/pubmed/23708665
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/onc.2013.172
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