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MK-2206 Causes Growth Suppression and Reduces Neuroendocrine Tumor Marker Production in Medullary Thyroid Cancer through Akt Inhibition

BACKGROUND: Development of targeted therapies for medullary thyroid cancer (MTC) has focused on inhibition of the RET (Rearranged during Transfection) proto-oncogene. Akt has been shown to be a downstream target of RET via the key mediator phosphoinositide-3-kinase. MK-2206 is an orally administered...

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Bibliografski detalji
Glavni autori: Burke, Jocelyn F., Schlosser, Logan, Harrison, April D., Kunnimalaiyaan, Muthusamy, Chen, Herbert
Format: Artigo
Jezik:Inglês
Izdano: 2013
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3955957/
https://ncbi.nlm.nih.gov/pubmed/23900743
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1245/s10434-013-3168-2
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