Oxidative activation of the Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) regulates vascular smooth muscle migration and apoptosis

Activation of the Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and reactive oxygen species (ROS) promote neointimal hyperplasia after vascular injury. CaMKII can be directly activated by ROS through oxidation. In this study, we determined whether abolishing the oxidative activation site of...

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Detalhes bibliográficos
Main Authors: Zhu, Linda J., Klutho, Paula J., Scott, Jason A., Xie, Litao, Luczak, Elizabeth D., Dibbern, Megan E., Prasad, Anand M., Jaffer, Omar A., Venema, Ashlee N., Nguyen, Emily K., Guan, Xiaoqun, Anderson, Mark E., Grumbach, Isabella M.
Formato: Artigo
Idioma:Inglês
Publicado em: 2014
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3955051/
https://ncbi.nlm.nih.gov/pubmed/24418021
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.vph.2014.01.001
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