Transforming Growth Factor-β(1) Represses Bone Morphogenetic Protein–Mediated Smad Signaling in Pulmonary Artery Smooth Muscle Cells via Smad3

Схожие документы

• Sex Affects Bone Morphogenetic Protein Type II Receptor Signaling in Pulmonary Artery Smooth Muscle Cells
  по: Mair, Kirsty M., et al.
  Опубликовано: (2015)
• Bone Morphogenetic Protein (BMP) and Activin Type II Receptors Balance BMP9 Signals Mediated by Activin Receptor-like Kinase-1 in Human Pulmonary Artery Endothelial Cells
  по: Upton, Paul D., et al.
  Опубликовано: (2009)
• Bone Morphogenetic Protein Receptor Type II Deficiency and Increased Inflammatory Cytokine Production. A Gateway to Pulmonary Arterial Hypertension
  по: Soon, Elaine, et al.
  Опубликовано: (2015)
• BMP type II receptor deficiency confers resistance to growth inhibition by TGF-β in pulmonary artery smooth muscle cells: role of proinflammatory cytokines
  по: Davies, Rachel J., et al.
  Опубликовано: (2012)
• Transforming Growth Factor β Inhibits Bone Morphogenetic Protein-Induced Transcription through Novel Phosphorylated Smad1/5-Smad3 Complexes
  по: Grönroos, Eva, et al.
  Опубликовано: (2012)