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Are therapeutic effects of antiacne agents mediated by activation of FoxO1 and inhibition of mTORC1?

Acne pathogenesis has recently been linked to decreased nuclear FoxO1 levels and increased mTORC1 activity. This hypothesis postulates that antiacne agents either enhance nuclear FoxO activity or inhibit mTORC1. Benzoyl peroxide (BPO), by activation of oxidative stress-inducible kinases, increases n...

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Détails bibliographiques
Auteurs principaux: Melnik, Bodo C, Schmitz, Gerd
Format: Artigo
Langue:Inglês
Publié: Blackwell Publishing Ltd 2013
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC3746104/
https://ncbi.nlm.nih.gov/pubmed/23800068
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/exd.12172
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