ATR phosphorylates SMARCAL1 to prevent replication fork collapse

The DNA damage response kinase ataxia telangiectasia and Rad3-related (ATR) coordinates much of the cellular response to replication stress. The exact mechanisms by which ATR regulates DNA synthesis in conditions of replication stress are largely unknown, but this activity is critical for the viabil...

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Bibliographische Detailangaben
Hauptverfasser: Couch, Frank B., Bansbach, Carol E., Driscoll, Robert, Luzwick, Jessica W., Glick, Gloria G., Bétous, Rémy, Carroll, Clinton M., Jung, Sung Yun, Qin, Jun, Cimprich, Karlene A., Cortez, David
Format: Artigo
Sprache:Inglês
Veröffentlicht: Cold Spring Harbor Laboratory Press 2013
Schlagworte:
Research Paper
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3731549/
https://ncbi.nlm.nih.gov/pubmed/23873943
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/gad.214080.113
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