Mechanism of the postreceptor defect in insulin action in human obesity. Decrease in glucose transport system activity.

We have studied insulin-stimulated 3-O-methyl glucose transport by isolated adipocytes prepared from 10 normal and 11 obese individuals. The results demonstrated that the insulin-glucose transport dose-response curves were shifted to the right in cells from the obese patients, and that the magnitude...

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Veröffentlicht in:	J Clin Invest
Hauptverfasser:	Ciaraldi, T P, Kolterman, O G, Olefsky, J M
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	American Society for Clinical Investigation 1981
Schlagworte:	Research Article
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC370874/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7026616/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI110342
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