Mechanism of the postreceptor defect in insulin action in human obesity. Decrease in glucose transport system activity.
We have studied insulin-stimulated 3-O-methyl glucose transport by isolated adipocytes prepared from 10 normal and 11 obese individuals. The results demonstrated that the insulin-glucose transport dose-response curves were shifted to the right in cells from the obese patients, and that the magnitude...
محفوظ في:
| الحاوية / القاعدة: | J Clin Invest |
|---|---|
| المؤلفون الرئيسيون: | , , |
| التنسيق: | Artigo |
| اللغة: | Inglês |
| منشور في: |
American Society for Clinical Investigation
1981
|
| الموضوعات: | |
| الوصول للمادة أونلاين: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC370874/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7026616/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI110342 |
| الوسوم: |
لا توجد وسوم, كن أول من يضع وسما على هذه التسجيلة!
|