Mechanism of the postreceptor defect in insulin action in human obesity. Decrease in glucose transport system activity.
We have studied insulin-stimulated 3-O-methyl glucose transport by isolated adipocytes prepared from 10 normal and 11 obese individuals. The results demonstrated that the insulin-glucose transport dose-response curves were shifted to the right in cells from the obese patients, and that the magnitude...
Gardado en:
| Publicado en: | J Clin Invest |
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| Principais autores: | , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
American Society for Clinical Investigation
1981
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC370874/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7026616/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI110342 |
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