Knockout of the Na,K-ATPase α(2)-isoform in cardiac myocytes delays pressure overload-induced cardiac dysfunction

Lignende værker

• Mice expressing ouabain-sensitive α1-Na,K-ATPase have increased susceptibility to pressure overload-induced cardiac hypertrophy
  af: Wansapura, Arshani N., et al.
  Udgivet: (2011)
• Knockout of the Na,K-ATPase α(2)-isoform in the cardiovascular system does not alter basal blood pressure but prevents ACTH-induced hypertension
  af: Rindler, Tara N., et al.
  Udgivet: (2011)
• Extracellular potassium dependence of the Na(+)-K(+)-ATPase in cardiac myocytes: isoform specificity and effect of phospholemman
  af: Han, Fei, et al.
  Udgivet: (2009)
• Na(+)/K(+)-ATPase α2-isoform preferentially modulates Ca(2+) transients and sarcoplasmic reticulum Ca(2+) release in cardiac myocytes
  af: Despa, Sanda, et al.
  Udgivet: (2012)
• Marinobufagenin enhances cardiac contractility in mice with ouabain-sensitive α(1) Na(+)-K(+)-ATPase
  af: Wansapura, Arshani N., et al.
  Udgivet: (2009)