Knockout of the Na,K-ATPase α(2)-isoform in the cardiovascular system does not alter basal blood pressure but prevents ACTH-induced hypertension

Lignende værker

• Knockout of the Na,K-ATPase α(2)-isoform in cardiac myocytes delays pressure overload-induced cardiac dysfunction
  af: Rindler, Tara N., et al.
  Udgivet: (2013)
• ACTH-induced hypertension is dependent on the ouabain-binding site of the α(2)-Na(+)-K(+)-ATPase subunit
  af: Lorenz, John N., et al.
  Udgivet: (2008)
• The Ouabain-Binding Site of the α2 Isoform of Na,K-ATPase Plays a Role in Blood Pressure Regulation During Pregnancy
  af: Oshiro, Naomi, et al.
  Udgivet: (2010)
• Hypertension from chronic central sodium chloride in mice is mediated by the ouabain-binding site on the Na,K-ATPase α(2)-isoform
  af: Van Huysse, James W., et al.
  Udgivet: (2011)
• The highly conserved cardiac glycoside binding site of Na,K-ATPase plays a role in blood pressure regulation
  af: Dostanic-Larson, Iva, et al.
  Udgivet: (2005)