Endothelial NOS (NOS3) impairs myocardial function in developing sepsis

Endothelial nitric oxide synthase (NOS)3-derived nitric oxide (NO) modulates inotropic response and diastolic interval for optimal cardiac performance under non-inflammatory conditions. In sepsis, excessive NO production plays a key role in severe hypotension and myocardial dysfunction. We aimed to...

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Bibliographic Details
Main Authors:	van de Sandt, Annette M., Windler, Rainer, Gödecke, Axel, Ohlig, Jan, Zander, Simone, Reinartz, Michael, Graf, Jürgen, van Faassen, Ernst E., Rassaf, Tienush, Schrader, Jürgen, Kelm, Malte, Merx, Marc W.
Format:	Artigo
Language:	Inglês
Published:	Springer-Verlag 2013
Subjects:	Original Contribution
Online Access:	https://ncbi.nlm.nih.gov/pmc/articles/PMC3597270/ https://ncbi.nlm.nih.gov/pubmed/23397596 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00395-013-0330-8
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Endothelial NOS (NOS3) impairs myocardial function in developing sepsis

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