Circulating NOS3 Modulates Left Ventricular Remodeling following Reperfused Myocardial Infarction

PURPOSE: Nitric oxide (NO) is constitutively produced and released from the endothelium and several blood cell types by the isoform 3 of the NO synthase (NOS3). We have shown that NO protects against myocardial ischemia/reperfusion (I/R) injury and that depletion of circulating NOS3 increases within...

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Pubblicato in:PLoS One
Autori principali: Gorressen, Simone, Stern, Manuel, van de Sandt, Annette M., Cortese-Krott, Miriam M., Ohlig, Jan, Rassaf, Tienush, Gödecke, Axel, Fischer, Jens W., Heusch, Gerd, Merx, Marc W., Kelm, Malte
Natura: Artigo
Lingua:Inglês
Pubblicazione: Public Library of Science 2015
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Research Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4397096/
https://ncbi.nlm.nih.gov/pubmed/25875863
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0120961
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