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Endothelial NOS (NOS3) impairs myocardial function in developing sepsis

Endothelial nitric oxide synthase (NOS)3-derived nitric oxide (NO) modulates inotropic response and diastolic interval for optimal cardiac performance under non-inflammatory conditions. In sepsis, excessive NO production plays a key role in severe hypotension and myocardial dysfunction. We aimed to...

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Xehetasun bibliografikoak
Egile Nagusiak: van de Sandt, Annette M., Windler, Rainer, Gödecke, Axel, Ohlig, Jan, Zander, Simone, Reinartz, Michael, Graf, Jürgen, van Faassen, Ernst E., Rassaf, Tienush, Schrader, Jürgen, Kelm, Malte, Merx, Marc W.
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: Springer-Verlag 2013
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC3597270/
https://ncbi.nlm.nih.gov/pubmed/23397596
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00395-013-0330-8
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