Prolonged apoptotic cell accumulation in germinal centers of Mer-deficient mice causes elevated B cell and CD4(+) helper T cell responses leading to autoantibody production

Lignende værker

• Impaired apoptotic cell clearance in the germinal center by Mer-deficient tingible body macrophages leads to enhanced antibody-forming cell and germinal center responses
  af: Rahman, Ziaur S.M., et al.
  Udgivet: (2010)
• B cell-intrinsic TLR7 signaling is essential for the development of spontaneous germinal centers
  af: Soni, Chetna, et al.
  Udgivet: (2014)
• B cell-intrinsic CD84 and Ly108 maintain germinal center B cell tolerance
  af: Wong, Eric B., et al.
  Udgivet: (2015)
• The lupus-prone NZM2410/NZW strain derived Sle1b sub-locus alters the germinal center checkpoint in female mice in a B cell intrinsic manner
  af: Wong, Eric B., et al.
  Udgivet: (2012)
• IFN-γ receptor and STAT1 signaling in B cells are central to spontaneous germinal center formation and autoimmunity
  af: Domeier, Phillip P., et al.
  Udgivet: (2016)