CaMKII determines mitochondrial stress responses in heart

Myocardial cell death is initiated by excessive mitochondrial Ca(2+) entry, causing Ca(2+) overload, mitochondrial permeability transition pore (mPTP) opening and dissipation of the mitochondrial inner membrane potential (ΔΨm)(1,2). However, the signaling pathways that control mitochondrial Ca(2+) e...

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Autors principals: Joiner, Mei-ling A., Koval, Olha M., Jingdong, Li, He, B. Julie, Allamargot, Chantal, Gao, Zhan, Luczak, Elizabeth D., Hall, Duane D., Fink, Brian D., Chen, Biyi, Yang, Jinying, Moore, Steven A., Scholz, Thomas D., Strack, Stefan, Mohler, Peter J., Sivitz, William I., Song, Long-Sheng, Anderson, Mark E.
Format: Artigo
Idioma:Inglês
Publicat: 2012
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Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3471377/
https://ncbi.nlm.nih.gov/pubmed/23051746
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nature11444
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