Oxidation of CaMKII determines cardiotoxic effects of aldosterone

Excessive activation of β-adrenergic, angiotensin II, and aldosterone (Aldo) signaling pathways promotes mortality after myocardial infarction (MI), while antagonist drugs targeting these pathways are core therapies for treating post-MI patients. Catecholamines and angiotensin II activate the multif...

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Detalhes bibliográficos
Main Authors: He, B. Julie, Joiner, Mei-Ling A., Singh, Madhu V., Luczak, Elizabeth D., Swaminathan, Paari Dominic, Koval, Olha M., Kutschke, William, Allamargot, Chantal, Yang, Jinying, Guan, Xiaoqun, Zimmerman, Kathy, Grumbach, Isabella M., Weiss, Robert M., Spitz, Douglas R., Sigmund, Curt D., Blankesteijn, W. Matthijs, Heymans, Stephane, Mohler, Peter J., Anderson, Mark E.
Formato: Artigo
Idioma:Inglês
Publicado em: 2011
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3332099/
https://ncbi.nlm.nih.gov/pubmed/22081025
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nm.2506
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