Oxidation of CaMKII determines cardiotoxic effects of aldosterone

Excessive activation of β-adrenergic, angiotensin II, and aldosterone (Aldo) signaling pathways promotes mortality after myocardial infarction (MI), while antagonist drugs targeting these pathways are core therapies for treating post-MI patients. Catecholamines and angiotensin II activate the multif...

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Bibliografiska uppgifter
Huvudupphovsmän: He, B. Julie, Joiner, Mei-Ling A., Singh, Madhu V., Luczak, Elizabeth D., Swaminathan, Paari Dominic, Koval, Olha M., Kutschke, William, Allamargot, Chantal, Yang, Jinying, Guan, Xiaoqun, Zimmerman, Kathy, Grumbach, Isabella M., Weiss, Robert M., Spitz, Douglas R., Sigmund, Curt D., Blankesteijn, W. Matthijs, Heymans, Stephane, Mohler, Peter J., Anderson, Mark E.
Materialtyp: Artigo
Språk:Inglês
Publicerad: 2011
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Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC3332099/
https://ncbi.nlm.nih.gov/pubmed/22081025
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nm.2506
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