Oxidation of CaMKII determines cardiotoxic effects of aldosterone

Excessive activation of β-adrenergic, angiotensin II, and aldosterone (Aldo) signaling pathways promotes mortality after myocardial infarction (MI), while antagonist drugs targeting these pathways are core therapies for treating post-MI patients. Catecholamines and angiotensin II activate the multif...

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Bibliografische gegevens
Hoofdauteurs: He, B. Julie, Joiner, Mei-Ling A., Singh, Madhu V., Luczak, Elizabeth D., Swaminathan, Paari Dominic, Koval, Olha M., Kutschke, William, Allamargot, Chantal, Yang, Jinying, Guan, Xiaoqun, Zimmerman, Kathy, Grumbach, Isabella M., Weiss, Robert M., Spitz, Douglas R., Sigmund, Curt D., Blankesteijn, W. Matthijs, Heymans, Stephane, Mohler, Peter J., Anderson, Mark E.
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: 2011
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Article
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3332099/
https://ncbi.nlm.nih.gov/pubmed/22081025
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nm.2506
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