Oxidation of CaMKII determines cardiotoxic effects of aldosterone

Excessive activation of β-adrenergic, angiotensin II, and aldosterone (Aldo) signaling pathways promotes mortality after myocardial infarction (MI), while antagonist drugs targeting these pathways are core therapies for treating post-MI patients. Catecholamines and angiotensin II activate the multif...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: He, B. Julie, Joiner, Mei-Ling A., Singh, Madhu V., Luczak, Elizabeth D., Swaminathan, Paari Dominic, Koval, Olha M., Kutschke, William, Allamargot, Chantal, Yang, Jinying, Guan, Xiaoqun, Zimmerman, Kathy, Grumbach, Isabella M., Weiss, Robert M., Spitz, Douglas R., Sigmund, Curt D., Blankesteijn, W. Matthijs, Heymans, Stephane, Mohler, Peter J., Anderson, Mark E.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2011
Aiheet:
Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3332099/
https://ncbi.nlm.nih.gov/pubmed/22081025
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nm.2506
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