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Blocking SCN10A channels in heart reduces late sodium current and is antiarrhythmic

RATIONALE: While the sodium channel locus SCN10A has been implicated by genome-wide association studies as a modulator of cardiac electrophysiology, the role of its gene product Nav1.8 as a modulator of cardiac ion currents is unknown. OBJECTIVE: We determined the electrophysiological and pharmacolo...

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Detalles Bibliográficos
Main Authors: Yang, Tao, Atack, Thomas C., Stroud, Dina Myers, Zhang, Wei, Hall, Lynn, Roden, Dan M.
Formato: Artigo
Idioma:Inglês
Publicado: 2012
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC3412150/
https://ncbi.nlm.nih.gov/pubmed/22723299
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.112.265173
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