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Targeting CDK1 promotes FLT3-activated acute myeloid leukemia differentiation through C/EBPα
Mutations that activate the fms-like tyrosine kinase 3 (FLT3) receptor are among the most prevalent mutations in acute myeloid leukemias. The oncogenic role of FLT3 mutants has been attributed to the abnormal activation of several downstream signaling pathways, such as STAT3, STAT5, ERK1/2, and AKT....
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Clinical Investigation
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3408728/ https://ncbi.nlm.nih.gov/pubmed/22797303 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI43354 |
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