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uPAR and cathepsin B inhibition enhanced radiation-induced apoptosis in gliomainitiating cells
Glioblastomas present as diffuse tumors with invasion into normal brain tissue and frequently recur or progress after radiation as focal masses because of glioma-initiating cells. The role of the urokinase-type plasminogen activator receptor (uPAR) and cathepsin B in stem-like phenotype has been ext...
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| Autori principali: | , , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Oxford University Press
2012
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3367854/ https://ncbi.nlm.nih.gov/pubmed/22573309 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/neuonc/nos088 |
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