Abnormal Termination of Ca(2+) Release Is a Common Defect of RyR2 Mutations Associated With Cardiomyopathies

Samankaltaisia teoksia

• Reduced threshold for store overload-induced Ca(2+) release is a common defect of RyR1 mutations associated with malignant hyperthermia and central core disease
  Tekijä: Chen, Wenqian, et al.
  Julkaistu: (2017)
• Inactivation of Ca2+ release channels (ryanodine receptors RyR1 and RyR2) with rapid steps in [Ca2+] and voltage.
  Tekijä: Laver, D R, et al.
  Julkaistu: (1998)
• Recruiting RyRs to Open in a Ca(2+) Release Unit: Single-RyR Gating Properties Make RyR Group Dynamics
  Tekijä: Gillespie, Dirk
  Julkaistu: (2020)
• CaMKII-dependent phosphorylation of RyR2 promotes targetable pathological RyR2 conformational shift
  Tekijä: Uchinoumi, Hitoshi, et al.
  Julkaistu: (2016)
• RyR2 mutations linked to ventricular tachycardia and sudden death reduce the threshold for store-overload-induced Ca(2+) release (SOICR)
  Tekijä: Jiang, Dawei, et al.
  Julkaistu: (2004)