Abnormal Termination of Ca(2+) Release Is a Common Defect of RyR2 Mutations Associated With Cardiomyopathies

समान संसाधन

• Reduced threshold for store overload-induced Ca(2+) release is a common defect of RyR1 mutations associated with malignant hyperthermia and central core disease
  द्वारा: Chen, Wenqian, और अन्य
  प्रकाशित: (2017)
• Inactivation of Ca2+ release channels (ryanodine receptors RyR1 and RyR2) with rapid steps in [Ca2+] and voltage.
  द्वारा: Laver, D R, और अन्य
  प्रकाशित: (1998)
• Recruiting RyRs to Open in a Ca(2+) Release Unit: Single-RyR Gating Properties Make RyR Group Dynamics
  द्वारा: Gillespie, Dirk
  प्रकाशित: (2020)
• RyR2 mutations linked to ventricular tachycardia and sudden death reduce the threshold for store-overload-induced Ca(2+) release (SOICR)
  द्वारा: Jiang, Dawei, और अन्य
  प्रकाशित: (2004)
• CaMKII-dependent phosphorylation of RyR2 promotes targetable pathological RyR2 conformational shift
  द्वारा: Uchinoumi, Hitoshi, और अन्य
  प्रकाशित: (2016)