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Alternatively-spliced extra domain A of fibronectin promotes acute inflammation and brain injury following cerebral ischemia in mice
BACKGROUND AND PURPOSE: The fibronectin isoform containing the alternatively-spliced extra domain A (EDA(+)-FN) is normally absent from the circulation, but plasma levels of EDA(+)-FN can become markedly elevated in several human pathological conditions associated with inflammation including ischemi...
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| Hauptverfasser: | , , , , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
2012
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3335936/ https://ncbi.nlm.nih.gov/pubmed/22363055 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/STROKEAHA.111.635516 |
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