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Phospholemman is a Negative Feed-Forward Regulator of Ca(2+) in β-Adrenergic Signaling, Accelerating β-Adrenergic Inotropy

Sympathetic stimulation enhances cardiac contractility by stimulating β-adrenergic signaling and protein kinase A (PKA). Recently, phospholemman (PLM) has emerged as an important PKA substrate capable of regulating cytosolic Ca(2+) transients. However, it remains unclear how PLM contributes to β-adr...

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Autores principales: Yang, Jason H., Saucerman, Jeffrey J.
Formato: Artigo
Lenguaje:Inglês
Publicado: 2012
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC3327824/
https://ncbi.nlm.nih.gov/pubmed/22289214
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2011.12.015
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