Transcription Factor GATA4 Is Activated but Not Required for Insulin-like Growth Factor 1 (IGF1)-induced Cardiac Hypertrophy

Ítems similars

• Gata4 is required for maintenance of postnatal cardiac function and protection from pressure overload-induced heart failure
  per: Bisping, Egbert, et al.
  Publicat: (2006)
• Phosphoinositide 3-kinase(p110α) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy
  per: McMullen, Julie R., et al.
  Publicat: (2003)
• Deletion of Ribosomal S6 Kinases Does Not Attenuate Pathological, Physiological, or Insulin-Like Growth Factor 1 Receptor-Phosphoinositide 3-Kinase-Induced Cardiac Hypertrophy
  per: McMullen, Julie R., et al.
  Publicat: (2004)
• Correction for McMullen et al., “Deletion of Ribosomal S6 Kinases Does Not Attenuate Pathological, Physiological, or Insulin-Like Growth Factor 1 Receptor–Phosphoinositide 3-Kinase-Induced Cardiac Hypertrophy”
  per: McMullen, Julie R., et al.
  Publicat: (2021)
• Early Remodelling of Perinuclear Ca(2+) Stores and Nucleoplasmic Ca(2+) Signalling During the Development of Hypertrophy and Heart Failure
  per: Ljubojevic, Senka, et al.
  Publicat: (2014)