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Repeated morphine treatment-mediated hyperalgesia, allodynia and spinal glial activation are blocked by co-administration of a selective cannabinoid receptor type-2 agonist

Spinal glial activation has been implicated in sustained morphine-mediated paradoxical pain sensitization. Since activation of glial CB2 cannabinoid receptors attenuates spinal glial activation in neuropathies, we hypothesized that CB2 agonists may also attenuate sustained morphine–mediated spinal g...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: Tumati, Suneeta, Largent-Milnes, Tally M, Keresztes, Attila, Ren, Jiyang, Roeske, William R, Vanderah, Todd W., Varga, Eva V
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2012
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3298577/
https://ncbi.nlm.nih.gov/pubmed/22285397
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jneuroim.2011.12.021
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