Group VIA PLA(2) (iPLA(2)β) Is Activated Upstream of p38 Mitogen-activated Protein Kinase (MAPK) in Pancreatic Islet β-Cell Signaling

Ítems similars

• Glucose Homeostasis, Insulin Secretion, and Islet Phospholipids in Mice that Overexpress iPLA(2)β in Pancreatic β-Cells and in iPLA(2)β-Null Mice
  per: Bao, Shunzhong, et al.
  Publicat: (2007)
• Evidence for Proteolytic Processing and Stimulated Organelle Redistribution of iPLA(2)β
  per: Song, Haowei, et al.
  Publicat: (2010)
• Mice deficient in Group VIB phospholipase A(2) (iPLA(2)γ) exhibit relative resistance to obesity and metabolic abnormalities induced by a Western diet
  per: Song, Haowei, et al.
  Publicat: (2010)
• Pancreatic Islets and Insulinoma Cells Express a Novel Isoform of Group VIA Phospholipase A(2) (iPLA(2)β) that Participates in Glucose-Stimulated Insulin Secretion and Is Not Produced by Alternate Splicing of the iPLA(2)β Transcript
  per: Ramanadham, Sasanka, et al.
  Publicat: (2003)
• Apoptosis of Insulin-Secreting Cells Induced by Endoplasmic Reticulum Stress Is Amplified by Overexpression of Group VIA Calcium-Independent Phospholipase A(2) (iPLA(2)β) and Suppressed by Inhibition of iPLA(2)β
  per: Ramanadham, Sasanka, et al.
  Publicat: (2004)