Mice deficient in Group VIB phospholipase A(2) (iPLA(2)γ) exhibit relative resistance to obesity and metabolic abnormalities induced by a Western diet

Benzer Materyaller

• Group VIA PLA(2) (iPLA(2)β) Is Activated Upstream of p38 Mitogen-activated Protein Kinase (MAPK) in Pancreatic Islet β-Cell Signaling
  Yazar:: Song, Haowei, ve diğerleri
  Baskı/Yayın Bilgisi: (2012)
• Group VIB Phospholipase A(2) Promotes Proliferation of INS-1 Insulinoma Cells and Attenuates Lipid Peroxidation and Apoptosis Induced by Inflammatory Cytokines and Oxidant Agents
  Yazar:: Bao, Shunzhong, ve diğerleri
  Baskı/Yayın Bilgisi: (2012)
• Group VIB Calcium-Independent Phospholipase A(2) (iPLA(2)γ) Regulates Platelet Activation, Hemostasis and Thrombosis in Mice
  Yazar:: Yoda, Emiko, ve diğerleri
  Baskı/Yayın Bilgisi: (2014)
• Apoptosis of Insulin-Secreting Cells Induced by Endoplasmic Reticulum Stress Is Amplified by Overexpression of Group VIA Calcium-Independent Phospholipase A(2) (iPLA(2)β) and Suppressed by Inhibition of iPLA(2)β
  Yazar:: Ramanadham, Sasanka, ve diğerleri
  Baskı/Yayın Bilgisi: (2004)
• Glucose Homeostasis, Insulin Secretion, and Islet Phospholipids in Mice that Overexpress iPLA(2)β in Pancreatic β-Cells and in iPLA(2)β-Null Mice
  Yazar:: Bao, Shunzhong, ve diğerleri
  Baskı/Yayın Bilgisi: (2007)