Glucose Homeostasis, Insulin Secretion, and Islet Phospholipids in Mice that Overexpress iPLA(2)β in Pancreatic β-Cells and in iPLA(2)β-Null Mice

Ítems similars

• Group VIA PLA(2) (iPLA(2)β) Is Activated Upstream of p38 Mitogen-activated Protein Kinase (MAPK) in Pancreatic Islet β-Cell Signaling
  per: Song, Haowei, et al.
  Publicat: (2012)
• Genetic modulation of islet β-cell iPLA(2)β expression provides evidence for its impact on β-cell apoptosis and autophagy
  per: Lei, Xiaoyong, et al.
  Publicat: (2013)
• Age-Related Changes in Bone Morphology Are Accelerated in Group VIA Phospholipase A(2) (iPLA(2)β)-Null Mice
  per: Ramanadham, Sasanka, et al.
  Publicat: (2008)
• Apoptosis of Insulin-Secreting Cells Induced by Endoplasmic Reticulum Stress Is Amplified by Overexpression of Group VIA Calcium-Independent Phospholipase A(2) (iPLA(2)β) and Suppressed by Inhibition of iPLA(2)β
  per: Ramanadham, Sasanka, et al.
  Publicat: (2004)
• β-Cell Calcium-Independent Group VIA Phospholipase A(2) (iPLA(2)β): Tracking iPLA(2)β Movements in Response to Stimulation With Insulin Secretagogues in INS-1 Cells
  per: Bao, Shunzhong, et al.
  Publicat: (2004)